<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0864-0300</journal-id>
<journal-title><![CDATA[Revista Cubana de Investigaciones Biomédicas]]></journal-title>
<abbrev-journal-title><![CDATA[Rev Cubana Invest Bioméd]]></abbrev-journal-title>
<issn>0864-0300</issn>
<publisher>
<publisher-name><![CDATA[ECIMED]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0864-03002020000300024</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Patogenia de las manifestaciones neurológicas asociadas al SARS-CoV-2]]></article-title>
<article-title xml:lang="en"><![CDATA[Pathogenesis of neurological manifestations associated to SARS-CoV-2]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Padrón-González]]></surname>
<given-names><![CDATA[Alexander Ariel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Dorta-Contreras]]></surname>
<given-names><![CDATA[Alberto Juan]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Ciencias Médicas de La Habana Instituto de Ciencias Básicas y Preclínicas &#8220;Victoria de Girón&#8221; ]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de Ciencias Médicas de La Habana Facultad de Ciencias Médicas &#8220;Miguel Enríquez&#8221; Laboratorio Central de Líquido Cefalorraquídeo (LABCEL)]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>09</month>
<year>2020</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>09</month>
<year>2020</year>
</pub-date>
<volume>39</volume>
<numero>3</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_arttext&amp;pid=S0864-03002020000300024&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_abstract&amp;pid=S0864-03002020000300024&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_pdf&amp;pid=S0864-03002020000300024&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN  Introducción: Los coronavirus infectan al ser humano y pueden causar manifestaciones neurológicas en individuos susceptibles.  Objetivo: Describir la patogenia de las manifestaciones neurológicas en pacientes con la COVID-19.  Estrategia de búsqueda y criterios de selección:  Se realizó una revisión bibliográfica empleando la bibliografía nacional e internacional actualizada. Se realizó la búsqueda en Google Académico, se consultaron artículos de libre acceso en las bases de datos Pubmed y SciELO, desde enero de 2014 hasta el 6 de mayo de 2020. Fueron seleccionados 51 artículos (6 en idioma español, 45 en inglés) y un libro de neuroinmunología. Se utilizaron los términos de búsqueda &#8220;COVID-19&#8221;, &#8220;coronavirus&#8221;, &#8220;SARS-CoV-2&#8221;, &#8220;manifestaciones neurológicas&#8221;, &#8220;sistema nervioso&#8221;, &#8220;patogénesis&#8221;, según el descriptor de Ciencias de la Salud (DeCS).  Análisis e integración de la información: El SARS-CoV-2 entra al sistema nervioso por la vía linfática, hematógena, transináptica retrógada, por diseminación local a través del etmoides o por disfunción de la barrera hematoencefálica. La patogenia puede ser por la acción directa del virus o inmunomediada. En la pandemia de COVID-19 se reportan pacientes con manifestaciones neurológicas centrales, periféricas y musculoesqueléticas. Los síntomas más frecuentes son los trastornos del gusto, el olfato, cefaleas, mialgias y mareos. En las formas graves se reportan meningitis, encefalitis, síndrome de Guillain-Barré, ictus y encefalopatías.  Conclusiones: El SARS-CoV-2 puede afectar al sistema nervioso central y periférico. Causa principalmente manifestaciones leves y transitorias, aunque pueden ocurrir complicaciones neurológicas. Los mecanismos patogénicos principales son el daño citopático directo o mecanismos indirectos debido a una respuesta inflamatoria.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT  Introduction: Coronaviruses infect humans and may cause neurological manifestations in susceptible individuals.  Objective: Describe the pathogenesis of neurological manifestations in patients with COVID-19.  Search strategy and selection criteria:  A review was conducted of national and international updated bibliography. The search was carried out in Google Scholar and open access papers were consulted in the databases PubMed and SciELO from January 2014 to 6 May 2020. A total 51 papers (6 in Spanish and 45 in English) and a book on neuroimmunology were selected. The search terms used were &#8220;COVID-19&#8221;, &#8220;coronavirus&#8221;, &#8220;SARS-CoV-2&#8221;, &#8220;neurological manifestations&#8221;, &#8220;nervous system&#8221; and &#8220;pathogenesis&#8221;, in compliance with the Health Sciences Descriptors (DeCS).  Data analysis and integration: SARS-CoV-2 enters the nervous system by lymphatic, hematogenous, transynaptic, retrograde routes, by local dissemination through the ethmoid, or by dysfunction of the hematoencephalic barrier. Pathogenesis may be due to direct action by the virus or immunomediated. During the COVID-19 pandemic patients have been reported with central, peripheral and musculoskeletal neurological manifestations. The most common symptoms are taste and smell disorders, headache, myalgia and dizziness. Meningitis, encephalitis, Guillain-Barré syndrome, stroke and encephalopathies have been reported in severe forms of the disease.  Conclusions: SARS-CoV-2 may affect the central and the peripheral nervous system. It mainly causes mild, transient manifestations, but neurological complications may also occur. The main pathogenic mechanisms are direct cytophatic damage or indirect mechanisms resulting from an inflammatory response.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[SARS-CoV-2]]></kwd>
<kwd lng="es"><![CDATA[COVID-19]]></kwd>
<kwd lng="es"><![CDATA[coronavirus]]></kwd>
<kwd lng="es"><![CDATA[manifestaciones neurológicas]]></kwd>
<kwd lng="es"><![CDATA[sistema nervioso]]></kwd>
<kwd lng="es"><![CDATA[patogenia]]></kwd>
<kwd lng="es"><![CDATA[patogénesis]]></kwd>
<kwd lng="en"><![CDATA[SARS-CoV-2]]></kwd>
<kwd lng="en"><![CDATA[COVID-19]]></kwd>
<kwd lng="en"><![CDATA[coronavirus]]></kwd>
<kwd lng="en"><![CDATA[neurological manifestations]]></kwd>
<kwd lng="en"><![CDATA[nervous system]]></kwd>
<kwd lng="en"><![CDATA[pathogenesis]]></kwd>
</kwd-group>
</article-meta>
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