<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2221-2434</journal-id>
<journal-title><![CDATA[Revista Finlay]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. Finlay]]></abbrev-journal-title>
<issn>2221-2434</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Ciencias Médicas de Cienfuegos. Centro Provincial de información de Ciencias Médicas]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2221-24342020000200170</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Influencia de los radicales libres en la génesis de la ateroesclerosis]]></article-title>
<article-title xml:lang="en"><![CDATA[Influence of Free Radicals in the Origin of Atherosclerosis]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández Guerra,]]></surname>
<given-names><![CDATA[Yoel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rodríguez Gómez,]]></surname>
<given-names><![CDATA[Alain]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Villafuerte Reinante,]]></surname>
<given-names><![CDATA[Janet]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Marrero Silva,]]></surname>
<given-names><![CDATA[Ignacio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Mora Hernández,]]></surname>
<given-names><![CDATA[Carlos Manuel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Ciencias Médicas  ]]></institution>
<addr-line><![CDATA[Cienfuegos ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Policlínico Área I  ]]></institution>
<addr-line><![CDATA[Cienfuegos ]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2020</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2020</year>
</pub-date>
<volume>10</volume>
<numero>2</numero>
<fpage>170</fpage>
<lpage>178</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_arttext&amp;pid=S2221-24342020000200170&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_abstract&amp;pid=S2221-24342020000200170&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_pdf&amp;pid=S2221-24342020000200170&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN Muchos de los componentes patológicos de las enfermedades cardiovasculares están estrechamente relacionados con una desregulación del metabolismo de las especies reactivas de oxígeno que propician la aparición del llamado estrés oxidativo. La oxidación de las lipoproteínas de baja densidad, la disfunción endotelial y los procesos inflamatorios están mediados en parte por el aumento en la producción celular de las especies reactivas de oxígeno. Con el objetivo de realizar una actualización sobre los últimos hallazgos que explican la influencia de los radicales libres en la génesis de la aterosclerosis, se realiza la presente revisión bibliográfica en la que se concluyó que el oxígeno constituye la principal fuente de formación de radicales libres, cuya acción es fundamental en el surgimiento de aterosclerosis. El incremento del estrés oxidativo favorece todas las fases del proceso aterotrombótico: adhesión y agregación plaquetaria, proliferación celular, peroxidación lipídica y proteica, daños irreversibles al ADN y a la membrana mitocondrial e inestabilización de la placa.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT Most of the pathologic components of oxidative stress of cardiovascular diseases are closely related with diregulation of metabolism of reactive species of oxygen, which favor the appearance of the so called oxidative stress. The oxidation of low density lipoproteins, endothelial dysfunction and inflammatory processes are mediated in some way by cellular production of the reactive oxygen species. This review is conducted aimed at revising the latest findings that explain the influence of free radicals in the origin of atherosclerosis. It is concluded that oxygen is the main source of free radical formation whose action is determinant for the genesis of atherosclerosis. The increase of oxidative stress favors atherothrombatic phases: platelet adhesion and aggregation, cell proliferation, lipid and protein perioxidation, irreversible damage in DNA and mitochondrial membrane and inestabilzation/desestabilization of the plaque.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[aterosclerosis]]></kwd>
<kwd lng="es"><![CDATA[radicales libres]]></kwd>
<kwd lng="es"><![CDATA[estrés oxidativo]]></kwd>
<kwd lng="en"><![CDATA[atherosclerosis]]></kwd>
<kwd lng="en"><![CDATA[free radicals]]></kwd>
<kwd lng="en"><![CDATA[oxidative stress]]></kwd>
</kwd-group>
</article-meta>
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