<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1029-3019</journal-id>
<journal-title><![CDATA[MEDISAN]]></journal-title>
<abbrev-journal-title><![CDATA[MEDISAN]]></abbrev-journal-title>
<issn>1029-3019</issn>
<publisher>
<publisher-name><![CDATA[Centro Provincial de Información de Ciencias Médicas]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1029-30192019000601092</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Influencia de la hipoxia como noxa en el origen temprano de la obesidad y sus complicaciones durante la vida intrauterina]]></article-title>
<article-title xml:lang="en"><![CDATA[Influence of hypoxia as noxa in the early origin of obesity and its complications during intra-uterine life]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Basain Valdés]]></surname>
<given-names><![CDATA[José María]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Valdés Alonso]]></surname>
<given-names><![CDATA[María del Carmen]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pérez Martínez]]></surname>
<given-names><![CDATA[Margarita]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pérez Gesen]]></surname>
<given-names><![CDATA[Cecilia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Policlínico Universitario Carlos Manuel Portuondo Lambert  ]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Hospital Pediátrico Docente Juan Manuel Márquez  ]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Instituto Nacional de Endocrinología Departamento de Endocrinología Pediátrica ]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2019</year>
</pub-date>
<volume>23</volume>
<numero>6</numero>
<fpage>1092</fpage>
<lpage>1103</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_arttext&amp;pid=S1029-30192019000601092&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_abstract&amp;pid=S1029-30192019000601092&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_pdf&amp;pid=S1029-30192019000601092&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN La hipoxia es un factor importante que regula el desarrollo placentario y estimula la invasión del trofoblasto y la diferenciación, la angiogénesis y la vasculogénesis. Cuando ocurre la fecundación, la hipoxia a la que está expuesta el blastocisto regula su crecimiento, a la vez que limita el número de células del trofoblasto y el desarrollo placentario posterior, lo cual es clave en el transporte de nutrientes y oxígeno al feto en desarrollo; sin embargo, la hipoxia crónica fetoplacentaria conduce a disfunción vascular placentaria y a la programación intrauterina de enfermedades vasculares y metabólicas, ya que regula, a largo plazo, la expresión de enzimas relacionadas con la vía L-arginina/óxido nítrico en células endoteliales de diferentes lechos vasculares, incluyendo la placenta. Teniendo en cuenta los planteamientos anteriores en la presente investigación se describen los efectos de la hipoxia como noxa durante la vida intrauterina y su influencia en el origen temprano de la obesidad y sus complicaciones.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT Hypoxia is an important factor that regulates the placental development and stimulates the invasion of trophoblast as well as differentiation, angiogenesis and vasculogenesis. At the moment of fertilization, hypoxia to which the blastocyte is exposed, regulates its growth, at the same time that it limits the number of trophoblast cells and the posterior placental development, that is essential in the transport of nutrients and oxygen to the developing fetus; however, chronic fetus-placental hypoxia leads to vascular placental dysfunction and to intra-uterine programming of vascular and metabolic diseases, since it regulates, at long term, the expression of enzymes related to the L-arginine/nitric oxide way in endothelial cells of different vascular beds, including placenta. Taking this into account the effects of hypoxia as noxa during intra-uterine life and its influence in the early origin of obesity and its complications are described in the present investigation.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[hipoxia]]></kwd>
<kwd lng="es"><![CDATA[noxa]]></kwd>
<kwd lng="es"><![CDATA[programación fetal in utero]]></kwd>
<kwd lng="en"><![CDATA[hypoxia]]></kwd>
<kwd lng="en"><![CDATA[noxa]]></kwd>
<kwd lng="en"><![CDATA[in uterus fetal programming]]></kwd>
</kwd-group>
</article-meta>
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