<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1682-0037</journal-id>
<journal-title><![CDATA[Revista Cubana de Angiología y Cirugía Vascular]]></journal-title>
<abbrev-journal-title><![CDATA[Rev Cubana Angiol Cir Vasc]]></abbrev-journal-title>
<issn>1682-0037</issn>
<publisher>
<publisher-name><![CDATA[Centro Nacional de Información de Ciencias MédicasEditorial Ciencias Médicas]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1682-00372020000200005</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Caracterización del estado inmunometabólico de los pacientes con diagnóstico de pie diabético]]></article-title>
<article-title xml:lang="en"><![CDATA[Characterization of the immunometabolic status of patients with diagnosis of diabetic foot]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Álvarez Hidalgo]]></surname>
<given-names><![CDATA[Roberto Jesús]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Escalante Padrón]]></surname>
<given-names><![CDATA[Odalys]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Álvarez Escalante]]></surname>
<given-names><![CDATA[Gretel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital Universitario &#8220;Manuel Ascunce Domenéch&#8221;  ]]></institution>
<addr-line><![CDATA[Camagüey ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de Ciencias Médicas &#8220;Dr. Carlos J. Finlay&#8221;  ]]></institution>
<addr-line><![CDATA[Camagüey ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Centro de Inmunología Molecular  ]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>08</month>
<year>2020</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>08</month>
<year>2020</year>
</pub-date>
<volume>21</volume>
<numero>2</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_arttext&amp;pid=S1682-00372020000200005&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_abstract&amp;pid=S1682-00372020000200005&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_pdf&amp;pid=S1682-00372020000200005&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN  Introducción:  El pie diabético es una alteración clínica inducida por la hiperglucemia mantenida, con o sin isquemia y previo traumatismo, lesión y/o ulceración del pie. La hiperglucemia favorece las infecciones, pero no potencia el crecimiento bacteriano, lo que sugiere la participación del sistema inmune en esta susceptibilidad.  Objetivo:  Caracterizar el estado inmunometabólico de los pacientes con diagnóstico pie diabético.  Métodos:  Se realizó un estudio descriptivo de corte transversal en 25 pacientes ingresados con diagnóstico de pie diabético, entre septiembre y diciembre de 2018 en el Hospital Universitario &#8220;Manuel Ascunce Domenéch&#8221;. Las variables estudiadas fueron: edad, sexo, formas clínicas de presentación del pie diabético, complementos 3 y 4, inmunoglobulinas G y A, y hemoglobina glucosilada. Se empleó la estadística descriptiva para el análisis de los datos.  Resultados:  El sexo femenino representó el 60 % y los pacientes con más de 70 años el 40 %. La inmunoglobulina G sérica se encontró disminuida en un 28 % de los pacientes con pie diabético y aumentada en otro 28 %. La hemoglobina glucosilada reflejó un desbalance en el 48 % de los casos. De los pacientes con descontrol metabólico, 8 (32 %) mostraron alteraciones de la inmunoglobulina G y 2 del componente C3.  Conclusiones:  Existió un grupo de pacientes con pie diabético que presentaron alteraciones inmunológicas variables y/o descontrol metabólico. Estos podrían beneficiarse con un manejo integral a partir del uso de inmunoterapia y la prevención de complicaciones infecciosas del pie diabético.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT  Introduction: Diabetic foot is a clinic alteration induced by persistent hyperglycemias, with or without ischemia and previous trauma, lesion and/or ulceration of the foot. Hyperglycemia favours infections, but it does not increase the bacterial growth which suggests the participation of the immune system in this sensitivity.  Objective: To characterize the immunometabolic status of patients with diagnosis of diabetic foot.  Methods: It was carried out a descriptive, cross-sectional study in 25 patients hospitalized with diagnosis of diabetic foot among September and December, 2018 in &#8220;Manuel Ascunce Domenech&#8221; University Hospital. The studied variables were: age, sex, clinical forms of diabetic foot, complement 3 and 4, G and A, immunoglobulins and glycosylated hemoglobine. It was used descriptive statistic for data analysis.  Results: Females represented the 60% and patients older than 70 years were the 40%. Seric G immunoglobulin was found as decreased in 28% of the patients with diabetic foot and increased in the 28%. Glycosylated hemoglobine presented unbalanced in 48% of the cases. From the patients with metabolic decontrol, 8 (32%) showed alterations of G immunoglobulin and 2 in the component C3.  Conclusions: There was a group of patients with diabetic foot who presented variable immunological alterations and/or metabolic decontrol. Those patients can benefit with an integral management using immunotherapy and the prevention of infectious complications of the diabetic foot.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[diabetes mellitus]]></kwd>
<kwd lng="es"><![CDATA[pie diabético]]></kwd>
<kwd lng="es"><![CDATA[estado inmune, control metabólico]]></kwd>
<kwd lng="en"><![CDATA[Diabetes mellitus]]></kwd>
<kwd lng="en"><![CDATA[diabetic foot]]></kwd>
<kwd lng="en"><![CDATA[immune status]]></kwd>
<kwd lng="en"><![CDATA[metabolic control]]></kwd>
</kwd-group>
</article-meta>
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