<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1727-897X</journal-id>
<journal-title><![CDATA[MediSur]]></journal-title>
<abbrev-journal-title><![CDATA[Medisur]]></abbrev-journal-title>
<issn>1727-897X</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Ciencias Médicas de Cienfuegos, Centro Provincial de Ciencias Médicas, Provincia de Cienfuegos.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1727-897X2020000100073</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Marcadores morfológicos de hipoxia en el riñón fetal con insuficiencia placentaria tratado con neuro-EPO: estudio en ratas]]></article-title>
<article-title xml:lang="en"><![CDATA[Morphological markers of hypoxia in the fetal kidney with placental insufficiency treated with neuro-EPO: study in rats]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Nuñez López]]></surname>
<given-names><![CDATA[Nínive]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Nova Bonet]]></surname>
<given-names><![CDATA[Yaima]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Suárez Aguiar]]></surname>
<given-names><![CDATA[Aida María]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Alonso Padilla]]></surname>
<given-names><![CDATA[Laymit]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Morgado Gamboa]]></surname>
<given-names><![CDATA[Yordanca]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López Marín]]></surname>
<given-names><![CDATA[Laura]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Ciencias Médicas de La Habana Instituto Superior de Ciencias Médicas Victoria de Girón ]]></institution>
<addr-line><![CDATA[La Habana La Habana]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Hospital Clínico Quirúrgico Joaquín Albarrán  ]]></institution>
<addr-line><![CDATA[La Habana La Habana]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>02</month>
<year>2020</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>02</month>
<year>2020</year>
</pub-date>
<volume>18</volume>
<numero>1</numero>
<fpage>73</fpage>
<lpage>81</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_arttext&amp;pid=S1727-897X2020000100073&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_abstract&amp;pid=S1727-897X2020000100073&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_pdf&amp;pid=S1727-897X2020000100073&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN  Fundamento:  la restricción del crecimiento intrauterino constituye una complicación del embarazo. Los recién nacidos con esta condición están expuestos a un mayor riesgo de morbimortalidad perinatal y postnatal.  Objetivo:  evaluar marcadores morfológicos de hipoxia en el desarrollo fetal y en el riñón, utilizando un modelo de insuficiencia placentaria tratado con eritropoyetina humana con bajo contenido de ácido siálico (neuro-Epo) en ratas.  Métodos:  se utilizaron tres grupos de ratas gestadas de la línea Wistar. Un grupo control (grupo I) y dos grupos experimentales (grupos II y III) con seis ratas cada uno. A las ratas de los grupos II y III se les realizó ligaduras de arterias uterinas en el día 16 de la gestación (E 16). Al grupo III desde E16 hasta E19 se le administró una dosis de 0,5 mg/kg/día de neuro-Epo por vía subcutánea y al grupo II se les administró placebo. En el día 20 de la gestación se pesaron los fetos y sus placentas. En el feto se midió la talla y los diámetros cefálicos. Las características morfométricas e histológicas en el riñón fetal se estudiaron con tinción de hematoxilina-eosina y PAS. Se realizó un análisis cualitativo histopatológico de sus tipos celulares.  Resultados:  los fetos con restricción del crecimiento intrauterino no mejoraron los marcadores de crecimiento. Se encontraron lesiones por hipoxia en el riñón fetal del grupo RCIU no tratado que mejoraron al administrar neuro-Epo.  Conclusiones:  la administración de neuro-Epo solo mostró efectos reparadores y protectores sobre alteraciones histológicas provocadas por la hipoxia en el riñón fetal.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT  Foundation:  intrauterine growth restriction constitutes a complication of pregnancy. Newborns with this condition are exposed to an increased risk of perinatal and postnatal morbidity and mortality.  Objective:  to evaluate morphological markers of hypoxia in fetal and kidney development, using a model of placental insufficiency treated with human erythropoietin with low sialic acid content (neuro-Epo) in rats.  Methods:  three groups of gestated rats from the Wistar line were used. A control group (group I) and two experimental groups (groups II and III) with six rats each. Rats of groups II and III had uterine artery ligation on day 16 of pregnancy (E 16). Group III from E16 to E19 was administered a dose of 0.5 mg / kg / day of neuro-Epo subcutaneously and group II was administered placebo. On the 20th day of gestation the fetuses and their placentas were weighed. The fetuses&#8217; size and cephalic diameters were measured. Morphometric and histological features in the fetal kidney were studied with hematoxylin-eosin staining and PAS. A qualitative histopathological analysis of their cell types was performed.  Results:  fetuses with intrauterine growth restriction did not improve growth markers. Hypoxia lesions were found in the fetal kidney of the untreated RCIU group that improved by administering neuro-Epo.  Conclusions:  the administration of neuro-Epo only showed reparative and protective effects on histological alterations caused by hypoxia in the fetal kidney.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[hipoxia]]></kwd>
<kwd lng="es"><![CDATA[hipoxia fetal]]></kwd>
<kwd lng="es"><![CDATA[riñón]]></kwd>
<kwd lng="es"><![CDATA[insuficiencia placentaria]]></kwd>
<kwd lng="es"><![CDATA[eritropoyetina]]></kwd>
<kwd lng="es"><![CDATA[ratas Wistar]]></kwd>
<kwd lng="en"><![CDATA[hypoxia]]></kwd>
<kwd lng="en"><![CDATA[fetal hypoxia]]></kwd>
<kwd lng="en"><![CDATA[kidney]]></kwd>
<kwd lng="en"><![CDATA[placental insufficiency]]></kwd>
<kwd lng="en"><![CDATA[erythropoietin]]></kwd>
<kwd lng="en"><![CDATA[rats]]></kwd>
<kwd lng="en"><![CDATA[Wistar]]></kwd>
</kwd-group>
</article-meta>
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