<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1729-519X</journal-id>
<journal-title><![CDATA[Revista Habanera de Ciencias Médicas]]></journal-title>
<abbrev-journal-title><![CDATA[Rev haban cienc méd]]></abbrev-journal-title>
<issn>1729-519X</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Ciencias Médicas de la Habana]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1729-519X2019000100030</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[La inflamación desde una perspectiva inmunológica: desafío a la Medicina en el siglo XXl]]></article-title>
<article-title xml:lang="en"><![CDATA[Inflammation from an immunologic perspective: a challenge to medicine in the 21st century]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[González-Costa]]></surname>
<given-names><![CDATA[Maricarmen]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[González]]></surname>
<given-names><![CDATA[Alexander Ariel Padrón]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Ciencias Médicas de La Habana Instituto de Ciencias Básicas y Preclínicas &#8220;Victoria de Girón&#8221; ]]></institution>
<addr-line><![CDATA[La Habana ]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>02</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>02</month>
<year>2019</year>
</pub-date>
<volume>18</volume>
<numero>1</numero>
<fpage>30</fpage>
<lpage>44</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_arttext&amp;pid=S1729-519X2019000100030&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_abstract&amp;pid=S1729-519X2019000100030&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.sld.cu/scielo.php?script=sci_pdf&amp;pid=S1729-519X2019000100030&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN  Introducción: La inflamación es una respuesta homeostática del organismo. Es uno de los principales motivos de consulta en Cuba y el mundo. Existe una percepción errónea de que es una entidad aislada y siempre patológica. Es un proceso dinámico, complejo, sistémico y multifactorial. Por eso constituye un reto el dilucidar los elementos, cambios tisulares que causa y cómo proceder en la clínica ante un cuadro inflamatorio.  Objetivo: Describir la inflamación, su clasificación, elementos involucrados y cambios sistémicos desde una perspectiva inmunológica.  Material y Métodos:  Se realizó una revisión sobre el tema empleando la bibliografía actualizada y luego se consultaron artículos de libre acceso en las bases de datos Pubmed y Scielo en el período de enero de 2013 a diciembre de 2018.  Desarrollo:  La inflamación puede clasificarse según el daño, tiempo o los efectores involucrados. Las principales moléculas son las citocinas como TNF-&#945;, IFN-&#947;, IL-1&#946;, IL-10, IL-6, TGF-&#946;. Participan células como los neutrófilos, mastocitos, macrófagos, linfocitos T y las del endotelio vascular. Durante el proceso inflamatorio se modifican las funciones de casi todos los sistemas de órganos. En ciertos tipos de inflamación, es la respuesta adaptativa quien origina y perpetúa el proceso inflamatorio.  Conclusiones: En la actualidad se desconocen los acontecimientos que desencadenan inflamación crónica y cómo ocurre el daño tisular. El mayor desafío consiste en dilucidar las causas y mecanismos inmunológicos que conllevan a las manifestaciones inflamatorias sistémicas que se manifiestan como enfermedades neurológicas, cardiovasculares y autoinmunes, entre otras.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT  Introduction:  Inflammation is a homeostatic response of the body. It is one of the main reasons for consultation in Cuba and throughout world. There is a misperception that it is an isolated and always pathological entity. It is a dynamic, complex, systemic, and multifactorial process. Therefore, it is a challenge to elucidate the elements and the tissue changes that it causes to establish the best way to improve the clinical practice related to an inflammatory process.  Objective:  To describe inflammation, its classification, elements involved, and systemic changes from an immunological perspective.  Material and Methods:  A review of the topic was made using the updated bibliography. Free-access articles were consulted in Pubmed and Scielo databases in the period from January 2013 to December 2018.  Development:  Inflammation can be classified according to the damage, time or effectors involved. The main molecules are the cytokines like TNF-&#945;, IFN-&#947;, IL-1&#946;, IL-10, IL-6, TGF-&#946;. Some cells participate such as neutrophils, mastocytes, macrophages, T-lymphocyes, and the vascular endothelium. During the inflammatory process, the functions of almost all organ systems are modified. It produces systemic changes that are observed in physiological processes such as pregnancy or aging. Sometimes inflammation triggers diseases such as cardiovascular and neurological ones, and cancer. The pathophysiological mechanisms have not been clarified.  Conclusions:  Currently, the events that trigger chronic inflammation and how tissue damage occurs are unknown. The biggest challenge is to elucidate the causes and immunological mechanisms that lead to inflammatory manifestations that are expressed as systemic neurological, cardiovascular and autoimmune diseases, among others.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Inflamación aguda]]></kwd>
<kwd lng="es"><![CDATA[inflamación crónica]]></kwd>
<kwd lng="es"><![CDATA[mediadores proinflamatorios]]></kwd>
<kwd lng="es"><![CDATA[sepsis]]></kwd>
<kwd lng="en"><![CDATA[Acute inflammation]]></kwd>
<kwd lng="en"><![CDATA[chronic inflammation]]></kwd>
<kwd lng="en"><![CDATA[proinflammatory mediators]]></kwd>
<kwd lng="en"><![CDATA[sepsis]]></kwd>
</kwd-group>
</article-meta>
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